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In this Review, we highlight the intriguing roles that these studies have revealed for ESX systems in bacterial survival and pathogenicity during infection with M. Furthermore, we discuss the diversity of ESX systems that has been described among mycobacteria and selected non-mycobacterial species. In the context of mycobacterial infection, caspase-independent cell death suggests the involvement of non-endogenous factors, such as bacterial effectors, in mediating host cell death.

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